Session

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms

PREDICTIVE CLINICAL AND IMAGING FEATURES OF HIGH-GRADE DYSPLASIA IN MUCINOUS CYSTIC NEOPLASMS OF THE PANCREAS
PREDICTIVE CLINICAL AND IMAGING FEATURES OF HIGH-GRADE DYSPLASIA IN MUCINOUS CYSTIC NEOPLASMS OF THE PANCREAS

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms  et al.

A NOVEL EUS-FNB DERIVED ORGANOID CO-CULTURE MODEL OF PANCREATIC CANCER
A NOVEL EUS-FNB DERIVED ORGANOID CO-CULTURE MODEL OF PANCREATIC CANCER

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms  et al.

ONCOGENIC GNAS AND KRAS SIGNALING COLLECTIVELY DRIVE DYSPLASIA IN AN IPSC-DERIVED ORGANOID MODEL FOR CYSTIC PANCREATIC NEOPLASIA
ONCOGENIC GNAS AND KRAS SIGNALING COLLECTIVELY DRIVE DYSPLASIA IN AN IPSC-DERIVED ORGANOID MODEL FOR CYSTIC PANCREATIC NEOPLASIA

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms  et al.

HFE H63D VARIANT PROMOTES EPITHELIAL TO MESENCHYMAL TRANSITION IN PANCREATIC CANCER IN CELL CYCLE DEPENDENT MANNER
HFE H63D VARIANT PROMOTES EPITHELIAL TO MESENCHYMAL TRANSITION IN PANCREATIC CANCER IN CELL CYCLE DEPENDENT MANNER

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms  et al.

SURVEILLANCE FOR PRESUMED BD-IPMN OF THE PANCREAS: STABILITY, SIZE, AND AGE IDENTIFY TARGETS FOR DISCONTINUATION
SURVEILLANCE FOR PRESUMED BD-IPMN OF THE PANCREAS: STABILITY, SIZE, AND AGE IDENTIFY TARGETS FOR DISCONTINUATION

Cystic neoplasia at the pancreas: From EUS to molecular mechanisms  et al.

HFE H63D VARIANT PROMOTES EPITHELIAL TO MESENCHYMAL TRANSITION IN PANCREATIC CANCER IN CELL CYCLE DEPENDENT MANNER
HFE H63D VARIANT PROMOTES EPITHELIAL TO MESENCHYMAL TRANSITION IN PANCREATIC CANCER IN CELL CYCLE DEPENDENT MANNER

A NOVEL EUS-FNB DERIVED ORGANOID CO-CULTURE MODEL OF PANCREATIC CANCER
A NOVEL EUS-FNB DERIVED ORGANOID CO-CULTURE MODEL OF PANCREATIC CANCER

ONCOGENIC GNAS AND KRAS SIGNALING COLLECTIVELY DRIVE DYSPLASIA IN AN IPSC-DERIVED ORGANOID MODEL FOR CYSTIC PANCREATIC NEOPLASIA
ONCOGENIC GNAS AND KRAS SIGNALING COLLECTIVELY DRIVE DYSPLASIA IN AN IPSC-DERIVED ORGANOID MODEL FOR CYSTIC PANCREATIC NEOPLASIA

PREDICTIVE CLINICAL AND IMAGING FEATURES OF HIGH-GRADE DYSPLASIA IN MUCINOUS CYSTIC NEOPLASMS OF THE PANCREAS
PREDICTIVE CLINICAL AND IMAGING FEATURES OF HIGH-GRADE DYSPLASIA IN MUCINOUS CYSTIC NEOPLASMS OF THE PANCREAS

SURVEILLANCE FOR PRESUMED BD-IPMN OF THE PANCREAS: STABILITY, SIZE, AND AGE IDENTIFY TARGETS FOR DISCONTINUATION
SURVEILLANCE FOR PRESUMED BD-IPMN OF THE PANCREAS: STABILITY, SIZE, AND AGE IDENTIFY TARGETS FOR DISCONTINUATION

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