Author

Marcin Magierowski

INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING
INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING

Marcin Magierowski

INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT'S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING
INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT'S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING

VOLUNTARY EXERCISE COMBINED WITH CONCOMITANT ALKALINE PHOSPHATASE TREATMENT RESOLVES EXPERIMENTAL COLITIS IN OBESE MICE BY IMPROVING COLONIC MICROCIRCULATION AND ATTENUATING INFLAMMATION, OXIDATIVE STRESS, AND RESTORATION OF ADIPONECTIN/LEPTIN RATIO
VOLUNTARY EXERCISE COMBINED WITH CONCOMITANT ALKALINE PHOSPHATASE TREATMENT RESOLVES EXPERIMENTAL COLITIS IN OBESE MICE BY IMPROVING COLONIC MICROCIRCULATION AND ATTENUATING INFLAMMATION, OXIDATIVE STRESS, AND RESTORATION OF ADIPONECTIN/LEPTIN RATIO

HYDROGEN SULFIDE (H2S)-RELEASING AP-39 MODULATES GASTRIC MUCOSAL MITOCHONDRIAL ACTIVITY PREVENTING THE OXIDATIVE ISCHEMIA/REPERFUSION (I/R)-INDUCED GASTRIC MUCOSAL INJURY
HYDROGEN SULFIDE (H2S)-RELEASING AP-39 MODULATES GASTRIC MUCOSAL MITOCHONDRIAL ACTIVITY PREVENTING THE OXIDATIVE ISCHEMIA/REPERFUSION (I/R)-INDUCED GASTRIC MUCOSAL INJURY

MITOCHONDRIA-TARGETING HYDROGEN SULFIDE-RELEASING AP-39 IN PREVENTION OF CHEMICALLY-INDUCED GASTRIC MUCOSAL DAMAGE
MITOCHONDRIA-TARGETING HYDROGEN SULFIDE-RELEASING AP-39 IN PREVENTION OF CHEMICALLY-INDUCED GASTRIC MUCOSAL DAMAGE

Marcin Magierowski

GASTROPROTECTIVE ACTION OF APELIN/AJP SYSTEM AGAINST ISCHEMIA-REPERFUSION INJURY. ROLE OF OXIDATIVE STRESS, NITRIC OXIDE AND SENSORY NEUROPEPTIDES
GASTROPROTECTIVE ACTION OF APELIN/AJP SYSTEM AGAINST ISCHEMIA-REPERFUSION INJURY. ROLE OF OXIDATIVE STRESS, NITRIC OXIDE AND SENSORY NEUROPEPTIDES

Marcin Magierowski  et al.

HYDROGEN SULFIDE SIGNALING IN GERD-INDUCED MTOR-DEPENDENT PALIGENOSIS: IMPLICATION FOR BARRETT'S ESOPHAGUS PATHOGENESIS
HYDROGEN SULFIDE SIGNALING IN GERD-INDUCED MTOR-DEPENDENT PALIGENOSIS: IMPLICATION FOR BARRETT'S ESOPHAGUS PATHOGENESIS

Marcin Magierowski  et al.

MATERNAL OBESITY INDUCED BY DIFFERENT DIET PROGRAM ALTERS COGNITIVE FUNCTIONS, THE PLASMA LEVELS OF APPETITE HORMONES AND INTESTINAL EXPRESSION OF ADIPOKINES AND ADIPO RECEPTORS 1 AND 2 IN MALE AND FEMALE RAT OFFSPRING
MATERNAL OBESITY INDUCED BY DIFFERENT DIET PROGRAM ALTERS COGNITIVE FUNCTIONS, THE PLASMA LEVELS OF APPETITE HORMONES AND INTESTINAL EXPRESSION OF ADIPOKINES AND ADIPO RECEPTORS 1 AND 2 IN MALE AND FEMALE RAT OFFSPRING

Marcin Magierowski  et al.

VOLUNTARY EXERCISE COMBINED WITH CONCOMITANT ALKALINE PHOSPHATASE TREATMENT RESOLVES EXPERIMENTAL COLITIS IN OBESE MICE BY IMPROVING COLONIC MICROCIRCULATION AND ATTENUATING INFLAMMATION, OXIDATIVE STRESS, AND RESTORATION OF ADIPONECTIN/LEPTIN RATIO
VOLUNTARY EXERCISE COMBINED WITH CONCOMITANT ALKALINE PHOSPHATASE TREATMENT RESOLVES EXPERIMENTAL COLITIS IN OBESE MICE BY IMPROVING COLONIC MICROCIRCULATION AND ATTENUATING INFLAMMATION, OXIDATIVE STRESS, AND RESTORATION OF ADIPONECTIN/LEPTIN RATIO

Marcin Magierowski  et al.

THE ROLE OF THE GUT MICROBIOTA AND EXPRESSION OF TIGHT JUNCTION PROTEINS IN ALLEVIATION BY INTESTINAL ALKALINE PHOSPHATASE TREATMENT OF EXPERIMENTAL COLITIS EXACERBATED IN HIGH-FAT-DIET FED MICE BY FORCED PHYSICAL ACTIVITY
THE ROLE OF THE GUT MICROBIOTA AND EXPRESSION OF TIGHT JUNCTION PROTEINS IN ALLEVIATION BY INTESTINAL ALKALINE PHOSPHATASE TREATMENT OF EXPERIMENTAL COLITIS EXACERBATED IN HIGH-FAT-DIET FED MICE BY FORCED PHYSICAL ACTIVITY

Marcin Magierowski  et al.

NOVEL INSIGHT INTO MECHANISM OF HEALING OF EXPERIMENTAL COLITIS BY INTESTINAL ALKALINE PHOSPHATASE IN OBESE MICE SUBJECTED TO FORCED TREADMILL EXERCISE. INVOLVEMENT OF OXIDATIVE STRESS, PROINFLAMMATORY BIOMARKERS AND INTESTINAL BARRIER PROTEINS
NOVEL INSIGHT INTO MECHANISM OF HEALING OF EXPERIMENTAL COLITIS BY INTESTINAL ALKALINE PHOSPHATASE IN OBESE MICE SUBJECTED TO FORCED TREADMILL EXERCISE. INVOLVEMENT OF OXIDATIVE STRESS, PROINFLAMMATORY BIOMARKERS AND INTESTINAL BARRIER PROTEINS

Marcin Magierowski  et al.

BENEFICIAL EFFECT OF INTESTINAL ALKALINE PHOSPHATASE IN THE MECHANISM OF MODULATION OF THE GENE EXPRESSION OF PROINFLAMMATORY BIOMARKERS IN THE ADIPOSE TISSUE, LIVER AND SKELETAL MUSCLE OF TREADMILL EXERCISING OBESE MICE WITH COLITIS
BENEFICIAL EFFECT OF INTESTINAL ALKALINE PHOSPHATASE IN THE MECHANISM OF MODULATION OF THE GENE EXPRESSION OF PROINFLAMMATORY BIOMARKERS IN THE ADIPOSE TISSUE, LIVER AND SKELETAL MUSCLE OF TREADMILL EXERCISING OBESE MICE WITH COLITIS

Marcin Magierowski  et al.

MITOCHONDRIA-TARGETING HYDROGEN SULFIDE-RELEASING AP-39 IN PREVENTION OF CHEMICALLY-INDUCED GASTRIC MUCOSAL DAMAGE
MITOCHONDRIA-TARGETING HYDROGEN SULFIDE-RELEASING AP-39 IN PREVENTION OF CHEMICALLY-INDUCED GASTRIC MUCOSAL DAMAGE

Marcin Magierowski  et al.

INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING
INHIBITORY EFFECT OF SLOW H2S-RELEASING DONOR (GYY4137) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND INFLAMMATORY SIGNALING

Marcin Magierowski  et al.

INHIBITORY EFFECT OF PHARMACOLOGICALLY-DERIVED OR ENDOGENOUSLY PRODUCED HYDROGEN SULFIDE (H2S) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA THROUGH IL-1BETA/COX-2/PGE2 PATHWAY LOOP
INHIBITORY EFFECT OF PHARMACOLOGICALLY-DERIVED OR ENDOGENOUSLY PRODUCED HYDROGEN SULFIDE (H2S) ON THE DEVELOPMENT OF BARRETT’S METAPLASIA THROUGH IL-1BETA/COX-2/PGE2 PATHWAY LOOP

Marcin Magierowski  et al.

HYDROGEN SULFIDE (H2S)-RELEASING AP-39 MODULATES GASTRIC MUCOSAL MITOCHONDRIAL ACTIVITY PREVENTING THE OXIDATIVE ISCHEMIA/REPERFUSION (I/R)-INDUCED GASTRIC MUCOSAL INJURY
HYDROGEN SULFIDE (H2S)-RELEASING AP-39 MODULATES GASTRIC MUCOSAL MITOCHONDRIAL ACTIVITY PREVENTING THE OXIDATIVE ISCHEMIA/REPERFUSION (I/R)-INDUCED GASTRIC MUCOSAL INJURY

Marcin Magierowski  et al.

CONTRIBUTION OF HYDROGEN SULFIDE-PRODUCING CYSTATHIONINE-G-LYASE (CTH) IN THE DEVELOPMENT OF BARRETT'S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND SYSTEMIC INFLAMMATION
CONTRIBUTION OF HYDROGEN SULFIDE-PRODUCING CYSTATHIONINE-G-LYASE (CTH) IN THE DEVELOPMENT OF BARRETT'S METAPLASIA-SPECIFIC MOLECULAR PROFILE AND SYSTEMIC INFLAMMATION

Marcin Magierowski  et al.

CONTRIBUTION OF ENZYMATIC ACTIVITY OF HEME OXYGENASE (HMOX) AND ITS BYPRODUCT, CARBON MONOXIDE (CO) IN BARRETT'S METAPLASIA DEVELOPMENT
CONTRIBUTION OF ENZYMATIC ACTIVITY OF HEME OXYGENASE (HMOX) AND ITS BYPRODUCT, CARBON MONOXIDE (CO) IN BARRETT'S METAPLASIA DEVELOPMENT

Marcin Magierowski  et al.

MITOCHONDRIA-TARGETED DELIVERY OF HYDROGEN SULFIDE (H2S)-PRODRUG COUNTERACTS GASTROTOXICITY OF NON-STEROIDAL ANTI-INFLAMMATORY DRUGS. INVOLVEMENT OF HEME OXYGENASE PATHWAY
MITOCHONDRIA-TARGETED DELIVERY OF HYDROGEN SULFIDE (H2S)-PRODRUG COUNTERACTS GASTROTOXICITY OF NON-STEROIDAL ANTI-INFLAMMATORY DRUGS. INVOLVEMENT OF HEME OXYGENASE PATHWAY

Marcin Magierowski  et al.

CONTROLLABLE DELIVERY OF CYTOPROTECTIVE CARBON MONOXIDE (CO) USING NOVEL CO-DELIVERING PRODRUGS COUNTERACTS BARRETT’S-LIKE METAPLASIA DEVELOPMENT
CONTROLLABLE DELIVERY OF CYTOPROTECTIVE CARBON MONOXIDE (CO) USING NOVEL CO-DELIVERING PRODRUGS COUNTERACTS BARRETT’S-LIKE METAPLASIA DEVELOPMENT

Marcin Magierowski  et al.

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